You are dispatched to the report of a sick case. Upon arrival, you find a 52-year old male
patient complaining of palpitations.
When pressed, he complains of very mild chest discomfort that he refuses
to describe as “pain,” but is worth a two on the 0-10 scale. He has an irregular pulse
rate of 130, a blood pressure of 116/70, and a respiratory rate of 16. He is warm, pink, and dry and is completely
awake and oriented with humor.
His ECG:
I started this post expecting to clear up misconceptions
about the differentiation of wide complex tachycardias (WCTs). I wanted to explain some of the main
differentiation algorhythms with their reported sensitivity and specificity. I was going to include algorhythms from
Brugada (1), the American College of Cardiology (2), Verecki (3), and Griffith
(4). I’ve taught 12-lead ECG
interpretation for more than 10 years, so this kind of topic is right in my
wheelhouse.
I decided not to waste my time or your time. The lesson is simpler than that: Prehospital and emergency differentiation of WCT is a waste of your time.
There are several issues with the differentiation of
WCTs. First, all of the algorhythms have
low specificity. Specificity is the
percentage of cases that are negative for the condition and are found to be
negative after applying the test – the true negative rate. So, low specificity means that the test
“pings” when the condition isn’t there.
Think of a radar detector in your windshield that alerts constantly, even when there is no speed
trap. Even the relatively higher
sensitivity (true positive) ratings of the WCT algorhythms are only in the high
80% range. That means you will get a
false positive (read: be on the wrong path) about 10% of the time.
The second issue is that the algorhythms are complex -
probably too complex for prehospital use, especially in front of a patient who
needs your time and full attention. For
example, the ACC algorhythm has four main decisions, plus 13 morphology clues
to remember. How often will you practice
using that? A sub-point of this one is
that most of the differentiation algorhythms involve precise measurements of R-wave
to peak times, nadirs, and Vi/Vt ratios* that are difficult to measure accurately.
The third reason not to differentiate in the field is that
there is no upside. Every decision you
make has a cost-benefit analysis, whether you overtly weigh the two sides or
not. The cost side doesn’t matter in
this case because there is no upside to getting the differentiation right - the
treatment (cardioversion or amiodarone) is the same for SVT and VT. And you won’t look as cool as you think you
will, like a paramedic Babe Ruth calling his shot. Anyone that hears your ECG interpretation will
assume that you just guessed right through chance, not skill.
The final reason is the most important: It does not matter!
Cardioversion works for supraventricular tachycardia with
aberrancy or with pre-existing bundle branch block. Cardioversion works for ventricular
tachycardia. Amiodarone works for
SVT. Amiodarone works for ventricular tachycardia. Adenosine can even convert ventricular
tachycardia on occasion! (5)
Your decisions should revolve around whether or not the
patient is stable or unstable. Unstable
patients get to ride the lightning – after a benzodiazepine, of course. For me, the decision to cardiovert someone
revolves around their level of responsiveness.
I am very hesitant to weld a patient who is awake, even with midazolam. But other medics have a lower threshold and
include chest pain as a sign of instability.
If the patient is stable, your next decision is pretty much
to think about adenosine, roll your eyes and skip the adenosine, and then start
an amiodarone drip. There are some
decisions that revolve around whether the presenting rhythm is regular or
irregular, but that decision is in the ACLS treatment algorhythm in order to
avoid giving adenosine (or other AV node blockers) to atrial fibrillation with
pre-excitation. You can avoid those
problems by just not
giving adenosine.
The differentiation of WCTs can be a fun skill to have. But you have to remember that it is a skill
applied after you have dropped the patient off at the hospital. It is a skill made for debating with your
partner at the ED dock or in the front of a bus at post. Not when you are in front of a patient.
Instead of studying the ECG that your monitor just printed
off, think about what else you could be doing.
Prioritize your actions. The
patient above, at the beginning of this post, has a pretty long list of
required actions. They include oxygen,
IV access, possible blood draw, a complete physical exam, a complete history of
the present illness, a complete health history, at least one 12-lead ECG,
aspirin, consideration of nitrates, a phone call to the receiving hospital so that they
are ready, considering/prepping an amiodarone infusion, repeat vital signs, constant monitoring, and extrication from wherever they
are to the ambulance. Pretty much all of
those would take higher priority than ten minutes of studying a field ECG with
a magnifying glass and a pair of calipers.
My heart-felt advice is to treat all wide complex
tachycardias as wide complex tachycardias, rather than trying to differentiate
whether it is a ventricular or supraventricular origin.
*Vi is the height/depth of the QRS after 40
milliseconds; Vt is the height/depth of the terminal 40
milliseconds. Want to measure those
while bouncing down the road, do you?
1. Brugada P, Brugada J, Mont L, Smeets J, Andries EW. A new
approach to the differential diagnosis of a regular tachycardia with a wide QRS
complex. Circulation. 1991; 83(5):
1649-1659.
2. Blomström-Lundqvist C, Scheinman MM, Aliot EM, Calkins H,
Camm AJ, et al. ACC/AHA/ESC guidelines for the management of patients with
supraventricular arrhythmias… J Am Coll
Cardiol. 2003; 42(8): 1493-1531.
3. Vereckei A, Duray G, Szénási G, Altemose GT, Miller JM.
Application of a new algorhythm in the differential diagnosis of wide QRS
tachycardia. Eur Heart J. 2007;
28(5): 589-600.
4. Griffith MJ, Garratt CJ, Mounsey P, Camm AJ. Ventricular
tachycardia as default diagnosis in broad complex tachycardia. Lancet. 1994; 343(8894): 386-388.
5. Marill KA, Wolfram S, Desouza IS, Nishijima DK, Kay D, Setnik GS, et al. Adenosine for wide-complex tachycardia: efficacy and safety. Crit Care Med. 2009; 37(9): 2512-2518.
5. Marill KA, Wolfram S, Desouza IS, Nishijima DK, Kay D, Setnik GS, et al. Adenosine for wide-complex tachycardia: efficacy and safety. Crit Care Med. 2009; 37(9): 2512-2518.